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Nutraceuticals
Quick Specs
FormFisetin flavonol
Purity≥98% (HPLC)
SourceRhus succedanea / semi-synthesis
Shelf life24 months
MOQ1 kg (sample); 5 kg (commercial)
Nutraceuticals

Fisetin

Flavonol Polyphenol ≥98% Purity Natural Senolytic SIRT1 Activator
The flavonoid that outperformed quercetin in a landmark Mayo Clinic study — nature's most potent senolytic, with SIRT1 activation, mTOR inhibition, and the strongest pre-clinical memory enhancement data of any polyphenol.
Fisetin (3,3',4',7-tetrahydroxyflavone) is a naturally occurring flavonol found in strawberries, apples, persimmons, onions, and cucumbers — with strawberries containing the highest concentration (~160 µg/g fresh weight). Commercially, fisetin is extracted from the dried fruits of Rhus succedanea (wax tree) or produced via semi-synthesis. CAS 528-48-3. Molecular formula C₁₅H₁₀O₆, MW 286.24 g/mol. Fisetin's structure is similar to quercetin but lacks a hydroxyl group at the C-5 position — this difference is significant because the 5-OH group in quercetin chelates iron and may limit certain bioactivities. Fisetin has a distinctive orange-yellow colour and is practically insoluble in water.

Fisetin catapulted to international attention in 2018 when a Mayo Clinic study published in EBioMedicine demonstrated it was the most potent senolytic of 10 flavonoids tested — more effective than quercetin at clearing senescent cells (zombie cells) and extending healthy lifespan in aged mice by 10% when administered late in life. It simultaneously activates SIRT1 (sirtuin-1) and inhibits mTOR, and has the most convincing pre-clinical memory enhancement data of any polyphenol, with multiple studies showing reversal of age-related cognitive decline in rodent models. SV Botanica supplies ≥98% fisetin from certified GMP/ISO facilities, tested by HPLC, light-protected packaging. The longevity supplement market is the primary demand driver with fisetin now a fixture in premium anti-aging stacks.
Available specifications
    Quality Assurance

    ≥98% HPLC-Verified. Senolytic-Grade Purity.

    Fisetin purity is confirmed by HPLC with specific quantification of fisetin aglycone — not total flavonoid content. Batch testing includes absence of fustin (the primary related compound), heavy metals, and microbial safety to GMP pharmacopoeia standards.

    01

    HPLC fisetin quantification

    COA reports specific fisetin aglycone content. Fustin (the 3-deoxyfisetin precursor in Rhus wood) is identified and reported separately as an impurity. Minimum 98% fisetin confirmed on every batch.

    02

    Light-protected packaging

    Fisetin's orange-yellow chromophore is photosensitive. Opaque, nitrogen-flushed aluminium packaging preserves both colour and bioactivity throughout storage and international transit.

    03

    Heavy metals & mycotoxin panel

    Botanical-source fisetin is tested for lead, mercury, arsenic, cadmium, ochratoxin A, and aflatoxins where applicable. Synthetic/semi-synthetic grades tested for residual solvents (ICH Q3C).

    04

    Export documentation

    COA, MSDS, Certificate of Origin, and Phytosanitary Certificate with every shipment. Halal and Kosher certificates on request. Full digital documentation before dispatch.

    Formulation Intelligence

    Fisetin Buyer's Guide: The Mayo Clinic Study, Senolytic Dosing & Formulation Considerations

    No natural compound has generated as much longevity science interest per dollar of consumer awareness as fisetin. Found in strawberries at microgram-per-gram levels, the idea that you would need to eat 37 strawberries per day to get a 100mg supplement dose helps consumers understand why concentrated bulk fisetin exists. But the science behind the 2018 Mayo Clinic results — and the translation from mouse studies to human protocols — requires careful interpretation for formulators building scientifically credible products.

    Landmark Study

    The 2018 Mayo Clinic Result

    CAS: 528-48-3

    Yousefzadeh et al., EBioMedicine 2018: fisetin was the most potent of 10 flavonoids tested as senolytics in aged mice. Administered late-in-life (month 22 of ~30 month lifespan), fisetin extended both median and maximum remaining lifespan. This is the most compelling longevity dataset for any natural flavonoid. Note: This was a mouse study — human trials on lifespan are impossible to conduct, but tissue senescence marker studies in humans are ongoing at Mayo.

    ✓ The gold-standard senolytic reference for fisetin product marketing.
    Natural Source

    Strawberries & Beyond

    Strawberry: 160 µg/g · Apple: 26 µg/g · Persimmon: 11 µg/g

    Natural fisetin content — Strawberry 160 µg/g; Apple 26 µg/g; Persimmon 11 µg/g; Onion 5 µg/g; Cucumber 0.1 µg/g. At 160 µg/g, a 100mg supplement dose requires approximately 625g of strawberries. This communication point resonates with consumers and validates the need for concentrated supplemental fisetin. Commercial extraction comes from Rhus succedanea wax tree fruits or semi-synthesis.

    Best positioning: "strawberry-sourced" for consumer supplement labels.
    Senolytic Protocol

    Pulse vs Daily Dosing

    Mouse equivalent: ~100 mg/kg · Human equiv.: ~500–1,000 mg

    The Mayo Clinic mouse study used continuous supplementation at ~100mg/kg. Translating this to human protocols, researchers are investigating both daily dosing (100–200mg) and intermittent high-dose "pulse" approaches (500–1,000mg for 2 consecutive days monthly). The pulse approach mimics the dasatinib/quercetin protocol logic. For consumer supplements, daily 100–200mg is most practical; clinical longevity products increasingly use the pulse protocol. No established human therapeutic dose yet — clinical trials ongoing.

    Note: No established human therapeutic dose yet — clinical trials ongoing.
    Combination Strategy

    Fisetin + Quercetin Stack

    Complementary senolytic mechanisms · PI3K/AKT + mast cell stabilisation

    Fisetin (most potent senolytic) + Quercetin (broadest anti-inflammatory + antiviral) is the most scientifically supported natural senolytic combination. Their mechanisms are complementary: fisetin inhibits PI3K/AKT survival pathways more potently; quercetin adds mast cell stabilisation and COMT inhibition. From a formulation standpoint, both flavonoids have poor oral bioavailability that can be addressed through phytosome complexation or fat co-ingestion.

    Fastest-growing segment of the senolytic supplement market.
    Buyer FAQ

    Frequently Asked Questions

    The designation comes from a landmark 2018 study published in EBioMedicine by researchers at the Mayo Clinic (Yousefzadeh et al.), which systematically tested 10 natural flavonoids for senolytic activity — the ability to selectively eliminate senescent "zombie" cells that accumulate with age and drive chronic inflammation. Fisetin was the clear winner: it reduced the proportion of highly elevated senescence markers in aged mouse tissues, reduced circulating inflammatory factors, and — critically — extended both median and maximum remaining lifespan when given to aged mice (24 months old, equivalent to ~75-year-old humans). Quercetin was the second most effective. The mechanism appears to involve inhibition of the PI3K/AKT survival pathways that senescent cells rely on disproportionately, along with reduction of the SASP (senescence-associated secretory phenotype) that makes senescent cells harmful.
    Both fisetin and quercetin have senolytic properties, but the 2018 Mayo Clinic study found fisetin significantly more potent than quercetin at clearing senescent cells from aged mouse tissues. In quantitative tissue analysis, fisetin reduced senescent cell markers (p16, p21, SA-β-gal) to a greater degree than quercetin at equivalent doses. However, quercetin has a much larger clinical literature base and is typically used in combination with dasatinib (a prescription drug) in the most-studied human senolytic protocol, while fisetin human trials are still emerging. In practice, longevity formulators increasingly use both together: fisetin for superior senolytic potency, quercetin for its broader anti-inflammatory and antiviral activity, and the combined presence of both compounds for product differentiation.
    Fisetin is the most extensively studied natural compound for cognitive function in preclinical models. Its memory-enhancing mechanisms include: (1) Promotion of long-term potentiation (LTP) in hippocampal neurons — LTP is the cellular mechanism underlying memory formation and consolidation; fisetin activates the ERK signalling pathway required for LTP induction; (2) SIRT1 activation in neural tissue — SIRT1 regulates BDNF (brain-derived neurotrophic factor) production, which supports synaptic plasticity and neurogenesis; (3) Reduction of amyloid-beta aggregation and tau protein phosphorylation — both hallmarks of Alzheimer's pathology; (4) Glutathione support — fisetin upregulates cysteine uptake in neurons via Nrf2 activation, increasing intracellular glutathione levels that protect against oxidative neurodegeneration. Human clinical trials on cognitive endpoints are ongoing.
    The 2018 Mayo Clinic senolytic study used doses equivalent to approximately 100mg/kg body weight in mice — translating allometrically to a human equivalent of approximately 500–1,000mg/day. However, most consumer supplements deliver 100–500mg fisetin per serving, as no established human therapeutic dose exists yet. Ongoing Mayo Clinic human clinical trials are using 20mg/kg body weight (~1,400mg for a 70kg person) in intermittent pulse protocols (2 consecutive days monthly), similar to the dasatinib/quercetin senolytic protocol. For general longevity and cognitive formulations without a specific senolytic protocol focus, 100–200mg daily is the most common consumer dosing range. Fisetin appears well-tolerated at these doses based on available safety data.
    Like most flavonoids, fisetin suffers from poor oral bioavailability due to low aqueous solubility and phase-II metabolism. Strategies to improve delivery include: (1) Fisetin Phytosome — complexing with phosphatidylcholine (similar to quercetin phytosome) significantly improves mucosal absorption; (2) Co-administration with fat — fisetin is lipid-soluble and absorption improves when taken with a meal containing fat; (3) Lipid-based delivery systems — soft gel capsules with oil excipients; (4) Nanoparticle or liposomal formulations for research-grade applications. For the senolytic pulse protocol (high dose, intermittent), taking fisetin with a fatty meal on the dosing days is the simplest practical recommendation. SV Botanica can supply fisetin phytosome complex for brands targeting enhanced bioavailability positioning.